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A positron emission tomography study of [18f]-florbetapir in Alzheimer's disease and frontotemporal dementia.
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J Neurol Neurosurg Psychiatry 2013 Nov; 84(11):e2
Authors: Christopher Kobylecki, Tobias Langheinrich, Rainer Hinz, Emma Rlc Vardy, Maria-Elena Martino, Anna Mt Richardson, Julie S Snowden, Jose Anton-Rodriguez, David Neary, David M Mann, Alex Gerhard, Karl Herholz
University of Manchester; Greater Manchester Neurosciences Centre; Newcastle University; CIBERSAN, Madrid, Spain.
Deposits of fibrillar amyloid ? (A?) protein are important in the pathophysiology of Alzheimer's disease (AD), but not in frontotemporal dementia (FTD). In vivo imaging of A? protein deposition, therefore, is a promising tool in the differential diagnosis of dementia syndromes. Previous pathological and imaging studies suggest that a minority of FTD patients may have coexistent A? deposition, possibly due to possession of the apolipoprotein E (APOE) ?4 allele. We report a PET study using the novel A? ligand [18F]-florbetapir in patients with AD, FTD and healthy controls.
PMID: 24109070 [PubMed - as supplied by publisher]